Fri Apr 25 10:49:57 SGT 2014  
    Hair loss, Singapore (SG)

Hair loss, Singapore (SG)


Hair loss, Singapore (SG) @singaporehair_com: Hair loss treatment clinic, Singapore


Advertisement: Come to sunny Singapore to have your testing and treatment. Singapore Ministry of Health registered general practice (GP) clinic:
168 Bedok South Avenue 3 #01-473
Singapore 460168
Tel: (+65) 6446 7446
Fax: (+65) 6449 7446
24hr Answering Tel: (+65) 6333 5550
Web: Hair loss, Singapore (SG)
Opening Hours
Monday to Friday: 9 am to 3 pm, 7 pm to 11 pm
Saturday & Sunday: 7 pm to 11 pm
Public Holidays: Closed
Last registration: one hour before closing time.
Walk-in clinic. Appointments not required.
Bring NRIC, Work Pass or Passport for registration.


Latest News

Common Dermatologic Conditions
Fri, 18 Apr 2014 21:36:27 +0100 | Medical Clinics of North America
This review discusses common dermatologic presentations as they would appear in a primary care office, exploring the differential diagnoses for each. Tips are provided on choosing an appropriate topical drug and vehicle and advising patients on its use. Etiology, differential diagnosis, and treatment options are discussed for the following: alopecias including androgenetic alopecia, female pattern hair loss, alopecia areata, and telogen effluvium; facial rashes including acne vulgaris, acne rosacea, periorificial dermatitis, seborrheic dermatitis, erysipelas/cellulitis, and systemic lupus erythematosus; intertriginous rashes including infections, intertrigo, and inverse psoriasis; and the inflamed leg including cellulitis and erysipelas, stasis dermatitis, and contact dermatitis. (Source: ...

Ceramide Synthase 4 deficiency in mice causes lipid alterations in sebum and results in alopecia
Thu, 17 Apr 2014 00:00:00 +0100 | BJ Disease
Five ceramide synthases (CerS2‑6) are expressed in mouse skin. While CerS3 has been shown to fulfill an essential function during skin development, neither CerS6 nor CerS2 deficient mice show an obvious skin phenotype. In order to study the role of CerS4, we generated CerS4 deficient mice (CerS4-/-) and CerS4 specific antibodies. With these biological tools we analyzed the tissue distribution and determined the cell type specific expression of CerS4 in suprabasal epidermal layers of footpads as well as in sebaceous glands of the dorsal skin. Loss of CerS4 protein leads to an altered lipid composition of the sebum, which is more solidified and therefore might cause the progressive hair loss due to physical blocking of the hair canal. We also noticed a strong decrease in C20 1,2&#x201...

Ceramide Synthase 4 deficiency in mice causes lipid alterations in sebum and results in alopecia.
Thu, 17 Apr 2014 00:00:00 +0100 | The Biochemical Journal
Authors: Ebel P, Imgrund S, Vom Dorp K, Hofmann K, Maier H, Drake H, Degen J, Dörmann P, Eckhardt M, Franz T, Willecke K

Male frontal fibrosing alopecia with generalised hair loss.
Wed, 16 Apr 2014 07:15:04 +0100 | The Australasian Journal of Dermatology
Authors: Chen W, Kigitsidou E, Prucha H, Ring J, Andres C

Frontal fibrosing alopecia: a retrospective clinical review of 62 patients with treatment outcome and long‐term follow‐up
Wed, 16 Apr 2014 00:00:00 +0100 | International Journal of Dermatology
ConclusionFrontal fibrosing alopecia is increasingly seen in postmenopausal women and rarely in men. Despite the limitations of a retrospective study, we conclude early intervention and treatment with intralesional triamcinolone acetonide may halt the progression of the disease; however, further controlled prospective studies are needed to establish treatment guidelines for frontal fibrosing alopecia. (Source: International Journal of Dermatology)

α‐Melanocyte‐stimulating hormone: a protective peptide against chemotherapy‐induced hair follicle damage?
Tue, 15 Apr 2014 00:00:00 +0100 | British Journal of Dermatology
ConclusionsExogenous α‐MSH exerts moderate HF‐protective effects against 4‐HC‐induced human scalp HF damage and upregulates the intrafollicular expression of a key cytoprotective enzyme. However, as substantial interindividual response variations were found, further studies are needed to probe α‐MSH as a candidate CIA‐protective agent. (Source: British Journal of Dermatology)

The oestrogen receptor 2 (ESR2) gene in female‐pattern hair loss: replication of association with rs10137185 in German patients
Tue, 15 Apr 2014 00:00:00 +0100 | British Journal of Dermatology
(Source: British Journal of Dermatology)

How Does Pediatric Psoriasis Present?
Mon, 14 Apr 2014 00:52:19 +0100 |

Meibomian gland dysfunction in a case of ichthyosis follicularis with alopecia and photophobia syndrome
Fri, 11 Apr 2014 00:00:00 +0100 | Indian Journal of Ophthalmology
Tarannum Fatima, Umang Mathur, Manisha AcharyaIndian Journal of Ophthalmology 2014 62(3):365-367We are reporting an interesting case of ichthyosis follicularis with alopecia and photophobia syndrome in a daughter and father from the Indian subcontinent associated with Meibomian gland dysfunction. A three-year-old female child presented with pain, redness and severe photophobia in both eyes since one month. Cutaneous examination revealed ichthyosis, absence of hair all over the body including eyebrows, scalp and angular cheilosis. Ocular examination revealed bilateral severe meibomianitis, multiple superficial punctate keratitides in right eye and corneal epithelial defect in the left eye. Other systemic features were prominent high forehead and large ears. Her father had similar symptoms. ...

Localized demodicosis due to Demodex cati on the muzzle of two cats treated with inhalant glucocorticoids.
Fri, 11 Apr 2014 00:00:00 +0100 | Veterinary Dermatology
CONCLUSIONS AND CLINICAL IMPORTANCE: Demodicosis should be considered as a possible differential diagnosis in cats with primary alopecia or other skin lesions on the face exposed to inhalant glucocorticoids. Minimization of contact between the inhalant glucocorticoid and the skin can be achieved by wiping residual powder from the face and by keeping the mask tightly pressed to the skin to avoid contact with the surrounding area.